AFI Super Curcumin C3 Complex w/ Bioperine

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Price: $35.95
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Suggested Use:   Take one caplet two to three times daily with food or as directed by your healthcare provider.

NOTE: This is a recommended substitute for those want the benefits of Curcumin C3 Complex (in Bosmeric-SR) but have a food sensitivity to ginger.  Also recommended to add AFI Boswellin AKBBA Max for the additional benefits of Boswellia to gain the similar benefits of Bosmeric-SR.

Curcumin C3 Complex® is a standardized extract from turmeric root (Curcuma longa) with a unique composition of curcuminoids (curcumin, demethoxycurcumin and bisdemethoxycurcumin). Curcumin C3 Complex® has been scientifically proven to be a powerful antioxidant preventing formation of free radicals and neutralizing already-formed free radicals, i.e., higher ORAC (Oxygen Radical Absorbance Capacity) value than green tea extract, green coffee extract and grape seed extract). Curcumin C3 Complex® is therefore considered to be a bio-protectant due to its dual activity of prevention and intervention of free radicals. Curcumin C3 Complex® may also relieve temporary joint inflammation and pain that occurs after overexertion during intensive physical activity. From a mode of action point of view, Arachidonic Acid is broken down via two major pathways (Arachidonic Acid Cascades). AA cascade via Cyclooxygenase, or COX pathway, generates inflammation-causing Prostaglandins and Thromboxanes, and AA cascade via Lipoxygenase, or LOX pathway, generates inflammation-causing Leukotrienes. Cyclooxygenase 2 (COX-2) becomes abundant in activated/stimulated macrophages and other cells at sites of inflammation. Curcuminoids naturally inhibit COX and LOX enzymes.†

BioPerine® is a patented natural extract from black pepper fruit which improves the absorption and bioavailability of nutrients. It has been shown to increase Curcumin C3 Complex by 2,000%.  The non-specific mechanisms for improving rapid absorption of nutrients may include increased blood supply to the GI tract, increased active nutrient transport by modulating efflux mechanisms (P-glycoprotein inhibition) and inhibition of solubilization by modulating metabolizing enzymes (glucuronidation).†